Tinnitus, Hyperacusis, and Misophonia Toolbox

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[DPOAE in tinnitus patients with cochlear hearing loss considering hyperacusis and misophonia].

UNLABELLED The most probable place generating tinnitus in auditory pathway are outer hair cells (OHC) inside cochlea. To asses their activity otoacoustic emission is used. The goal of the investigation was estimation the features of otoemission DPOAE in groups with tinnitus patients with cochlear hearing loss, estimation of diagnostic value of DPOAE parameters for analysis of function of the co...

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Treatments for Decreased Sound Tolerance (Hyperacusis and Misophonia)

Decreased sound tolerance (DST) is an underappreciated condition that affects the lives of a significant portion of the general population. There is lack of agreement regarding definitions, specific components, prevalence, methods of evaluation, and methods of treatment. Limited data are available on the results of treatments. Research is scant and constrained by the lack of an animal model. Th...

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Components of Decreased Sound Tolerance : Hyperacusis, Misophonia, Phonophobia

This report is an attempt to bring to your attention some important information related to decreased sound tolerance and it is based on a short paper (Jastreboff, MM and Jastreboff PJ, Hyperacusis, Audiology Online, June 2001, http://www.audiologyonline.com). In particular, as we recently proposed a new term “misophonia” and we would like to provide some explanation and justification for it (Ja...

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Pathology of Tinnitus and Hyperacusis-Clinical Implications

Not long ago, tinnitus and hyperacusis were considered intractable symptoms and the lack of interest and shortage of research in diseases with these symptoms would have made publishing a special issue on tinnitus and hyperacusis nearly impossible. During the past two decades, there has been an explosion of research on tinnitus and incremental growth on hyperacusis, a condition associated with h...

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Central Gain Control in Tinnitus and Hyperacusis

Sensorineural hearing loss induced by noise or ototoxic drug exposure reduces the neural activity transmitted from the cochlea to the central auditory system. Despite a reduced cochlear output, neural activity from more central auditory structures is paradoxically enhanced at suprathreshold intensities. This compensatory increase in the central auditory activity in response to the loss of senso...

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ژورنال

عنوان ژورنال: Seminars in Hearing

سال: 2014

ISSN: 0734-0451,1098-8955

DOI: 10.1055/s-0034-1372528